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Proteasome Inhibitor Immunotherapy for the Epithelial to Mesenchymal Transition: Assessing the A549 Lung Cancer Cell Microenvironment and the Role of M1, M2a and M2c ‘Hydrocortisone-Polarised’ Macrophages

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dc.contributor.authorSelin Engür
dc.contributor.authorDikmen, Miriş
dc.contributor.authorMiriş Dikmen
dc.contributor.orcid0000-0003-1534-8117
dc.contributor.orcid0009-0000-1090-0166
dc.date.accessioned2025-11-13T19:38:35Z
dc.date.issued2022-01-03
dc.identifier.doihttps://doi.org/10.21203/rs.3.rs-1068010/v1
dc.identifier.openalexW4205323107
dc.identifier.urihttps://hdl.handle.net/11421/11426
dc.identifier.urihttps://doi.org/10.21203/rs.3.rs-1068010/v1
dc.language.isoen
dc.relation.ispartofResearch Square (Research Square)
dc.rightsopenAccess
dc.subjectImmunotherapy
dc.subjectProteasome
dc.subjectLung cancer
dc.subjectTumor microenvironment
dc.subjectCancer research
dc.subjectMedicine
dc.subjectMesenchymal stem cell
dc.subjectEpithelial–mesenchymal transition
dc.subjectLung
dc.subjectCancer
dc.subjectPathology
dc.subjectChemistry
dc.subjectInternal medicine
dc.subjectTumor cells
dc.subjectMetastasis
dc.subjectBiochemistry
dc.subject.sdg3
dc.titleProteasome Inhibitor Immunotherapy for the Epithelial to Mesenchymal Transition: Assessing the A549 Lung Cancer Cell Microenvironment and the Role of M1, M2a and M2c ‘Hydrocortisone-Polarised’ Macrophages
dc.typepreprint
dspace.entity.typePublication
local.authorid.openalexA5042645402

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